A THEORY OF HEART DISEASE...
Pauling Therapy Explained
Well, I don't know that there is a need for a
randomized prospective, double-blind controlled trial when you get
evidence of this sort, the value of large intakes of vitamin C and
also of lysine for preventing the deposition of atherosclerotic
plaques, and preventing death from cardiovascular disease."
- Linus Pauling
You Must Unlearn What You Have Learned
 That atherosclerotic plaques deposit in response
to injury is accepted. The confusion in the media is cause and
effect. The fallacy is that cholesterol causes heart disease, but
plaque build-ups are the effect of heart disease. Our understanding
of the arterial healing process comes, in part, from research that
led to the 1985 Nobel Prize in Medicine.
 Cholesterol cannot and does not cause heart disease.
Human beings are well protected from scurvy by the RDA of vitamin
C (now 75-90 mg), but this meager amount virtually guarantees "hardening
of the arteries," i.e. the development of occlusive cardiovascular
disease as scabs (atherosclerotic plaques) form on the arterial
walls weakened by the vitamin deficiency.
 This realization is not new: G. C. Willis, MD, made the crucial
observation inthe early 1950s. A Canadian doctor, he noticed that
atherosclerotic plaques always formed in the same places. Usually
near the heart where the blood vessels are stretched and bent.
Willis suspected a vitamin C deficiency, and his experiments implicated
mechanical stress caused by the heart beat.
 The Pauling and Rath theory relies on the Willis observations.
(Note: In a heart bypass, veins from the leg are used which are
without plaque.) Because plaque does not form randomly throughout
the blood stream, it is unlikely that the primary cause of the
lesions leading to heart disease are "poisons" circulating
in the blood.
 We now know that plaques form over stress fractures. Visualize
stepping on a garden hose 70-80 times per minute, a fate similar
to the coronary arteries feeding the heart. Over time, human arteries
may wear down and develop small cracks. Mechanical stress then,
not cholesterol, causes heart disease. But why are humans more
susceptible to this stress than other beings with heart beats? Some
factor must cause the lesions in the walls of human blood vessels
but not in the coronary arteries of most other animals. Pauling
and Rath blame the lack of a specific protein caused by a specific
vitamin deficiency. A vitamin deficiency that is impossible
in most animals!
Roger J. Williams, PhD, in his 1971 book Nutrition Against Disease
explained vitamin C's role in collagen:
"Vitamin C is essential for the building
of collagen, the most abundant protein built in our bodies and the
major component of connective tissue. This connective tissue has
structural and supportive functions which are indispensable to heart
tissues, to blood vessels, --in fact, to all tissues. Collagen is
not only the most abundant protein in our bodies, it also occurs
in larger amounts than all other proteins put together. It cannot
be built without vitamin C. No heart or blood vessel or other organ
could possibly perform its functions without collagen. No heart
or blood vessel can be maintained in healthy condition without vitamin
Heart Disease *is* Chronic Scurvy
The Pauling and Rath theory postulates that the root cause of
atherosclerotic plaque deposits is a vitamin C deficiency.
 Pauling called this condition "chronic scurvy." It
is normal in humans, but it cannot happen in most other species.
Ascorbic acid (vitamin C) is not a vitamin for most animals in the
sense that it is not required in their diet. They make it in high
amounts in their livers or kidneys. The sad fact is that we humans
must obtain all our vitamin C from what we eat. (Unless we
supplement, our diets contain less than 1/100th of what animals
make, and we lose some in the gut during digestion.) According
to the Pauling/Rath theory, suboptimal vitamin C results in less
collagen. As collagen supplies dwindle, our blood vessels deteriorate.
In an acute shortage, we die of scurvy. In a chronic shortage, humans
develop atherosclerotic plaques. Pauling believed that most human
beings suffer chronic scurvy.
"Vitamin C has been under investigation,
reported in thousands of scientific papers, ever since it was discovered
(circa) fifty years ago. Even though some physicians had observed
forty or fifty years ago that amounts a hundred to a thousand times
larger (than the RDA) have value in controlling various diseases,the
medical profession and most scientists ignored this evidence."
Linus Pauling HOW TO LIVE LONGER AND FEEL BETTER,
pg 106 paperback.
 A vast amount of experimental research supports the Pauling/Rath
view. Careful studies with animals that do not make their own endogenous
vitamin C (such animals are rare) prove that when the dietary intake
of the vitamin is low, collagen production is limited, and blood
vessels tend to become thinner and weaker from wear and tear; plaque
deposits then form to compensate for this weakness. [Pauling/Rath,
87] Large population studies show that higher C intake results in
lower incidence of cardiovascular disease and lower death rates.[Enstrom,
Plaque is a Healing Response
 Plaque forms over injured blood vessels. If one suffers
plaque deposits, it is likely he/she owes his life to this material
that narrows arteries. Without plaque, the weakened blood vessels
would rupture or leak causing internal bleeding and death. A slower
version of scurvy, the disease long-dreaded by ancient sailors.
(James Lind discovered (year 1753) that eating fruit prevents
this disease. Acute scurvy can be prevented by a mere 10 mg vitamin
C per day. )
The correct terminology for cardiovascular (heart) disease then
is "chronic" scurvy or "sub clinical" scurvy.
 The human body's healing response to chronic scurvy is what
medicine calls coronary heart disease (CHD), AKA cardiovascular
disease (CVD), "heart disease", "atherosclerosis",
"arteriosclerosis", "hardening", "plaque",
"narrowing", etc. This process by itself rarely kills
people, but plaque lined arteries make heart attack more likely
from a blood clot or blockage. (Plaque lined arteries cannot easily
dilate in response to a clot.) Currently, it is unknown what amount
of vitamin C prevents the atherosclerotic plaques of chronic scurvy,
but Linus Pauling often recommended 3000 mg.
***Take Our Quiz***
Lp(a): The Surrogate Healing Factor
 The chronic scurvy healing process begins with an important
"sticky" form of cholesterol. Pauling and Rath were among
the first to attach utmost importance to the blood lipid: lipoprotein(a),
or Lp(a) for short. From the research that led to the 1985
Nobel prize, medical researchers learned how plaque deposits as
Lp(a) binds to lysine strands that appear in the arterial
walls. Lysine and proline are building blocks of the collagen super-molecule,
but the Cholesterol or Lysine strands the Lp(a) Binding Sites adhered
to are not normally exposed. The binding sites can adhere only after
blood vessels crack or suffer a small sore or lesion. Scientists
have since discovered the Lp(a) Proline Binding Sites
too. Note: Mainstream medical science has known since 1989 that
Lp(a) binds to form plaque, not ordinary LDL. [2, 4]
 Many experts believe that something circulating in the blood
must cause these cracks in our blood "pipes". For many
years, ordinary LDL cholesterol has been blamed because elevated
levels have sometimes been correlated with heart disease. Other
scientists correlated elevated homocysteine and oxidized cholesterol.
Again, the confusion is cause and effect. If cholesterol causes
cracks or lesions, plaque should be more randomly distributed throughout
the blood stream. According to the Pauling/Rath unified theory,
both elevated homocysteine and oxidized cholesterol are symptoms
New Oxford meta-analysis of
27 large studies (09/04/2000) shows that people with high Lp(a)
are 70% more likely to have a heart attack or stroke.
Plus: Only AMA publication to mention the Pauling protocol!
 Before teaming with Pauling, Dr. Rath's German research team
examined plaque from human aortas (blood vessels near the heart)
post-mortem. They discovered that atherosclerotic plaques are composed
primarily of Lp(a), not ordinary LDL cholesterol. [2, 4]
Dr. Rath, realized that Lp(a) was connected somehow with
vitamin C and joined the Linus Pauling Institute of Science and
Medicine. Together, Pauling and Rath developed their unified theory
which holds that increased Lp(a) acts as a surrogate for
low vitamin C and hardens weak blood vessels. Their experiments,
to test their theory, proved that low vitamin C intake will increase
blood levels of Lp(a) in test animals compared to controls.
 An important finding is that this sticky Lp(a) (a form
of cholesterol similar to LDL) has only been found in the very few
animal species that do not make their own vitamin C, including humans.
Today, most animals:
- Make vitamin C in their livers or kidneys, in large "mega"
amounts (9,000 mg to 12,000 mg adjusted for body weight - which
is high by current medical standards),
- do not have Lp(a) in their blood, and
- rarely suffer cardiovascular disease.
Humans are almost unique among life on Earth in that we must
receive our vitamin C entirely from the diet.
Endogenouns Vitamin C
Lp(a) in Blood
|High order Primates
|Other 99.9+% Species
Genetically engineered mice: Proof of the Pauling/Rath theory?
MEDICAL PARADIGM SHIFTING Vitamins-as-Prevention
 Linus Pauling the Chemist led the medical paradigm shift from
vitamins-as-prevention to vitamins-as-therapy. Pauling became fascinated
by vitamin C and other life-giving substances that, like drugs,
have powerful physiological effects in minuscule amounts, but unlike
most drugs, are completely non-toxic at very high dosages. His efforts
have failed to sway most medical doctors.
 In 1991, armed with the knowledge of the how and why Lp(a)
binds to our arteries creating plaque, Pauling invented the way
to unbind, or "turn off", the sticky Lp(a) molecule:
The cure for heart disease. The Pauling invention nullifies
the binding effect of Lp(a) to the damaged arterial wall.
The agents can be taken by mouth and act chemically as solvents
that both prevent and attack existing plaque formations. Importantly,
the formula attacks the root cause by stimulating the production
of collagen. With collagen, blood vessels stay healthy or heal normally,
so there are no cholesterol binding sites to attract Lp(a).
 According to one Pauling/Rath 1994 United States patent, the
amino acid lysine (lysine analogs), along with vitamin
C and other antioxidants (e.g. Co-Q10, vitamin E and
vitamin A), can, in sufficient concentration, inhibit Lp(a)
binding to exposed lysine residues. Proline residues are also exposed
by lesions in blood vessels; later experiments showed that proline
is a powerful binding inhibitor. Proline and lysine, with vitamin
C, other amino acids and antioxidants, in oral amounts well past
the amounts needed for prevention, become solvents by inhibiting
the binding of Lp(a). An Lp(a) binding inhibitor,
augmented with vitamin C, can stop and apparently even reverses
plaque formations. (Pauling and Rath's second U. S. patent is for
using binding inhibitors as solvents to melt atherosclerotic plaques
during organ transplants or heart surgery. The organ or blood vessel
is dipped in the Lp(a) Binding Inhibitor solution and any surface
plaque melts away. Also, arginine and proline have been shown to
inhibit the binding of apo(a) to the LDL cholesterol molecule, thus
interfering with the formation of Lp(a).)
 High intakes of these substances, esp. vitamin C and lysine,
are the Pauling Therapy. The Pauling Therapy treats the root-cause
and our seven-year experience demonstrates that at sufficient dosage,
these substances have powerful effects, and will rapidly reverse
advanced heart disease.
 The Pauling mega-vitamin/amino acid therapy increases blood
concentrations of important substances to:
- Strengthen and heal blood vessels,
- Lower Lp(a) blood levels and keep Lp(a) levels low, and
- Inhibit the binding of Lp(a) molecules to the walls of blood
The components of the Pauling Therapy are remarkably safe. Unlike
ordinary drugs, there are few known health risks. There are no known
adverse side effects, other than the laxative effect of vitamin
C. (We now believe that it is prudent to balance
high-dosage lysine with a good B-complex vitamin, and later during
maintenance, high-dose arginine.)
 Unlike the $2300/month cancer cure, the Pauling heart therapy
costs less than $100 per month. (Call us at 800-894-9025 for information
on the cure for cancer.)
Pharmaceutical Cartel's Nightmare
CURE FOR NUMBER ONE KILLER IS NOT A DRUG!
national ads credit pharmaceuticals for decrease
in Heart Disease mortality since 1970!?
 The Pauling "mega" dose ranges are based upon the
degree of illness in a patient. In general, Lp(a) binding
inhibitors are food substances that are non-toxic, and studies have
shown they improve health as intake increases; lesser amounts will
have lesser effects. According to his daughter Linda, Linus Pauling's
lysine dosage recommendations were carefully considered, and were
based on his knowledge of lysine blood serum levels, after intake.
It is not practical to obtain these amounts in food alone; supplements
 Vitamin C, and the amino acids lysine and proline, are the
fundamental building blocks of collagen and the Pauling Therapy
provides these building blocks in ample amounts. Over time, collagen
must be replenished for blood vessels to remain healthy and plaque
 The reason most doctors cannot accept these arguments is that
most medical schools do not teach that humans use up vitamin C while
producing collagen. This fact has been established. [See HOW TO
LIVE LONGER AND FEEL BETTER, Linus Pauling, 1986] One reason Linus
Pauling himself consumed 18,000 mg of vitamin C daily is that the
human ability to produce collagen is severely limited at the US
Recommended Daily Allowance (RDA) of 60 mg (now 75-90 mg)
Full text of this article see: